Publikationen von Lars Wittler
Alle Typen
Zeitschriftenartikel (45)
21.
Zeitschriftenartikel
20 (6), S. 599 - 607 (2018)
Noncoding copy-number variations are associated with congenital limb malformation. GENETICS IN MEDICINE 22.
Zeitschriftenartikel
50 (5), S. 662 - 667 (2018)
Polymer physics predicts the effects of structural variants on chromatin architecture. Nature Genetics 23.
Zeitschriftenartikel
19 (1), S. 118 - 134 (2018)
BRACHYURY directs histone acetylation to target loci during mesoderm development. EMBO Reports 24.
Zeitschriftenartikel
49 (10), S. 1539 - 1545 (2017)
Composition and dosage of a multipartite enhancer cluster control developmental expression of Ihh (Indian hedgehog). Nature Genetics 25.
Zeitschriftenartikel
42, S. 514 - 526 (2017)
Antagonistic Activities of Sox2 and Brachyury Control the Fate Choice of Neuro-Mesodermal Progenitors. Developmental Cell 26.
Zeitschriftenartikel
6 (6), S. 752 - 764 (2017)
Pattering and gastrulation deffects caused by the tw18 lethal are due to loss of Ppp2r1a. Biology Open 27.
Zeitschriftenartikel
538 (7624), S. 265 - 269 (2016)
Formation of novel chromatin domains determines pathogenicity of genomic duplications. Nature 28.
Zeitschriftenartikel
34 (7), S. 1790 - 800 (2016)
Different Concentrations of FGF Ligands, FGF2 or FGF8 Determine Distinct States of WNT-Induced Presomitic Mesoderm. Stem Cells 29.
Zeitschriftenartikel
26 (2), S. 183 - 191 (2016)
Exome Sequencing and CRISPR/Cas Genome Editing Identify Mutations of ZAK as a Cause of Limb Defects in Humans and Mice. Genome Research 30.
Zeitschriftenartikel
575 (2 Pt 2), S. 438 - 451 (2016)
Analysis of the Fam181 gene family during mouse development reveals distinct strain-specific expression patterns, suggesting a role in nervous system development and function. Gene 31.
Zeitschriftenartikel
161 (5), S. 1012 - 1025 (2015)
Disruptions of Topological Chromatin Domains Cause Pathogenic Rewiring of Gene-Enhancer Interactions. Cell 32.
Zeitschriftenartikel
10 (5), S. 833 - 839 (2015)
Deletions, Inversions, Duplications: Engineering of Structural Variants using CRISPR/Cas in Mice. Cell Reports 33.
Zeitschriftenartikel
23 (20), S. 5536 - 5544 (2014)
Genome-wide association study and mouse expression data identify a highly conserved 32 kb intergenic region between WNT3 and WNT9b as possible susceptibility locus for isolated classic exstrophy of the bladder. Human Molecular Genetics 34.
Zeitschriftenartikel
133, S. 23 - 35 (2014)
SRF is essential for mesodermal cell migration during elongation of the embryonic body axis. Mechanisms of Development 35.
Zeitschriftenartikel
100 (6), S. 512 - 517 (2014)
Classic bladder exstrophy: Frequent 22q11.21 duplications and definition of a 414 kb phenocritical region. Birth Defects Research Part A: Clinical and Molecular Teratology 36.
Zeitschriftenartikel
141 (11), S. 2325 - 2330 (2014)
The tissue-specific transcriptomic landscape of the mid-gestational mouse embryo. Development 37.
Zeitschriftenartikel
343 (6176), S. 1264 - 1266 (2014)
Dlk1 promotes a fast motor neuron biophysical signature required for peak force execution. Science 38.
Zeitschriftenartikel
21 (12), S. 1377 - 1382 (2013)
De novo microduplications at 1q41, 2q37.3, and 8q24.3 in patients with VATER/VACTERL association. European journal of human genetics: EJHG ; the official journal of the European Society of Human Genetics 39.
Zeitschriftenartikel
24 (2), S. 206 - 214 (2013)
The tissue-specific IncRNA Fendrr is an essential regulator of heart and body wall development in the mouse. Developmental Cell 40.
Zeitschriftenartikel
372 (1), S. 55 - 67 (2012)
In vivo knockdown of Brachyury results in skeletal defects and urorectal malformations resembling caudal regression syndrome. Developmental Biology